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As an interventional cardiologist in the UK, what do the results from the ODYSSEY Outcomes Trial and price reductions mean to the clinician at the front lines of cardiology care as far as access to and triggers for deploying a PCSK9 inhibitor?

As an interventional cardiologist in the UK, what do the results from the ODYSSEY Outcomes Trial and price reductions mean to the clinician at the front lines of cardiology care as far as access to and triggers for deploying a PCSK9 inhibitor?

How exactly did you and your colleagues model the pharmacoeconomic effectiveness of alirocumab based on the actual results of the ODYSSEY Outcomes Trial?

How exactly did you and your colleagues model the pharmacoeconomic effectiveness of alirocumab based on the actual results of the ODYSSEY Outcomes Trial?

What has been your clinical experience with respect to the comparative toleration of statins vs. PCSK9 inhibitors?

What has been your clinical experience with respect to the comparative toleration of statins vs. PCSK9 inhibitors?

In a high-risk patient who has undergone multiple stent procedures, what criteria do you prioritize for initiating a PCSK9 inhibitor in the setting of PCI, and what absolute LDL-C targets are in your cross-hairs based on the ODYSSEY Outcomes Trial?

In a high-risk patient who has undergone multiple stent procedures, what criteria do you prioritize for initiating a PCSK9 inhibitor in the setting of PCI, and what absolute LDL-C targets are in your cross-hairs based on the ODYSSEY Outcomes Trial?

Since ESC and ACC/AHA are examining hard LDL-C targets for patients at risk for ASCVD, how does ODYSSEY Outcomes help inform cardiologists about the rationale for PCSK9-mediated lowering to achieve ultra-low levels (i.e. <50 mg/dL) of LDL-C?

Since ESC and ACC/AHA are examining hard LDL-C targets for patients at risk for ASCVD, how does ODYSSEY Outcomes help inform cardiologists about the rationale for PCSK9-mediated lowering to achieve ultra-low levels (i.e. <50 mg/dL) of LDL-C?

Can you discuss the ESC Guidelines for LDL-C targets and the recent AHA Guidelines designating a target threshold of 70 mg/DL? Is this low enough? What is your recommendation for PCSK9 inhibition in the high-risk ACS patient who has undergone PCI?

Can you discuss the ESC Guidelines for LDL-C targets and the recent AHA Guidelines designating a target threshold of 70 mg/DL? Is this low enough? What is your recommendation for PCSK9 inhibition in the high-risk ACS patient who has undergone PCI?

You have identified the challenges of accessing PCSK9 inhibitors for patients whose CV risk is genetic vs. those whose clinical course is characterized by progressive vascular events. How do you approach each subset with respect to LDL-C management?

You have identified the challenges of accessing PCSK9 inhibitors for patients whose CV risk is genetic vs. those whose clinical course is characterized by progressive vascular events. How do you approach each subset with respect to LDL-C management?

Based on the ODYSSEY Outcomes and FOURIER Trials, which patient populations, including individuals with T2D, do you believe should be targeted for PCSK9 inhibitors?

Based on the ODYSSEY Outcomes and FOURIER Trials, which patient populations, including individuals with T2D, do you believe should be targeted for PCSK9 inhibitors?

As the Director of the Center for Cardiovascular Disease Prevention, what patient types on the risk landscape of ASCVD do you believe deserve special attention because they are likely to be eligible candidates for PCSK9-mediated LDL-C reduction?

As the Director of the Center for Cardiovascular Disease Prevention, what patient types on the risk landscape of ASCVD do you believe deserve special attention because they are likely to be eligible candidates for PCSK9-mediated LDL-C reduction?

What did ODYSSEY Outcomes and FOURIER teach us about high-risk patients with diabetes and the relative favorability of their responses to LDL-C lowering with PCSK9 inhibitors?

What did ODYSSEY Outcomes and FOURIER teach us about high-risk patients with diabetes and the relative favorability of their responses to LDL-C lowering with PCSK9 inhibitors?

Although the AHA Guidelines emphasize CV risk and mortality reductions with statins, ODYSSEY Outcomes provides comparable evidence for alirocumab. How compelling is the evidence for PCSK9 inhibition as a mediator of CV risk and mortality reduction?

Although the AHA Guidelines emphasize CV risk and mortality reductions with statins, ODYSSEY Outcomes provides comparable evidence for alirocumab. How compelling is the evidence for PCSK9 inhibition as a mediator of CV risk and mortality reduction?

With respect to patients with HeFH and homozygous FH who are challenged with lifelong CV disease risk burden due to genetic risk, what role do you believe PCSK9 inhibitors should play, especially in younger patient populations?

With respect to patients with HeFH and homozygous FH who are challenged with lifelong CV disease risk burden due to genetic risk, what role do you believe PCSK9 inhibitors should play, especially in younger patient populations?

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